Feifei Yi: Plant Physiology First Author

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Feifei Yi, first author of “Strigolactones positively regulate Verticillium wilt resistance in cotton through crosstalk with other hormone signaling pathways”

Current Position: Doctoral candidate at the State Key Laboratory of Cotton Biology, Henan University, Kaifeng, China

Education: Ph.D. in State Key Laboratory of Cotton Biology (Henan University, Kaifeng, China), M.S in Biotechnology Research Center (Southwest University, Chongqing, China), B.S. in School of Life Sciences (Henan Agriculture University, Zhengzhou, China)

Non-scientific Interests: Basketball, watching NBA games, photography, making video with my son

Brief bio: I developed my passion for plant sciences during my undergraduate studies at Henan Agriculture University. My interest on understanding the molecular mechanisms of plant fungal disease resistance was established during my master’s course at Southwest University, Chongqing. In this paper, we studied the function of plant hormones strigolactones (SLs) on cotton Verticillium wilt resistance and revealed the molecular mechanisms. Our results showed that SLs positively regulate cotton resistance to Verticillium wilt through crosstalk with the jasmonic acid (JA) and abscisic acid (ABA) -signaling pathways and by inducing reactive oxygen species (ROS) accumulation. Synchronously, the crosstalk between SLs and JA signaling pathway component, MYC2, might contribute to maintain the SLs homeostasis through a negative feedback loop. The candidate genes that resistance to Verticillium wilt in hormones pathway providing a foundational resource to design new crops that can survive plant biotic stresses.

姓名：易飞飞

现在职位：河南大学 棉花生物学国家重点实验室  2018级博士研究生

教育经历：

博士，植物学，2018-2023，河南大学

硕士，生物化学与分子生物学，2015-2018，西南大学

学士，生物技术，2011-2015，河南农业大学

兴趣：打篮球，摄影，听音乐，看电影

个人简介：

我本科毕业于河南农业大学，硕士毕业于西南大学生物化学与分子生物学专业，毕业后考入河南大学棉花生物学国家重点实验室，并攻读博士学位。在蔡应繁教授和李伟强研究员的指导下，开展了独脚金内酯（SLs）调控棉花黄萎病抗性中的功能及机制研究。通过SLs激素的外源施加及其合成的抑制、基因过表达和沉默、转录组分析、分子生物学等实验研究，发现了SLs正向参与棉花黄萎病抗性的功能并探究了其分子机制：1）SLs正调控棉花黄萎病的抗性，其合成基因GbCCD7和GbCCD8b的过表达增强了棉花黄萎病的抗性；2）SL正调控茉莉酸（JA）和脱落酸（ABA）信号通路相关防御基因的表达；3）JA信号途径转录因子GbMYC2与GbCCD7和GbCCD8b形成负反馈调节环调控棉花黄萎病的抗病性及维持激素间的稳态平衡，进而提高棉花在应对黄萎病菌入侵时的防御能力。未来，我们期望建立更加完善的激素间信号转导和增强黄萎病抗性的调控网络，更加精细化解读SLs在植物抗病中调控机制，为增强作物抗病能力提供理论依据和候选基因。