Jie Dong, first author of TCP4 and PIF3 antagonistically regulate organ-specific light induction of SAUR16/50 to modulate cotyledon opening during de-etiolation in Arabidopsis
Current Position: Postdoctoral Associate of Molecular, Cellular, and Developmental Biology, Yale University, USA
Education: Ph.D and B.S. in Biology, School of Life Sciences, Peking University, China
Non-scientific Interests: Badminton, watching movies, and cooking
Brief bio: I am mainly interested in how light signal shapes plant development using Arabidopsis as model. As a graduate student of Peking University, I had the great fortune of joining Prof. Xing Wang Deng lab from where I started my research career in 2012. In his lab, I have investigated how PIF protein stability is regulated by DET1 in the dark, and how light signal induces PIF3 protein degradation via EBF proteins after light exposure. After I got my Ph.D degree, I was lucky to join Dr. Ning Wei’s group at Yale University as a postdoctoral associate in 2016. At my current position, I elucidated the molecular basis of light induced cotyledon opening process of Arabidopsis seedling. Our study has revealed that a group of cotyledon-specific TCP transcription factors and the light responsive PIF transcription factors antagonistically modulate the expression of SAUR genes to regulate cotyledon opening of Arabidopsis seedling during dark to light transition.