Li Qin: The Plant Cell First Author
Li Qin, first author of “The ARP2/3 complex, acting cooperatively with Class I formins, modulates penetration resistance in Arabidopsis against powdery mildew invasion”
Current Position: Postdoctoral Research Associate in the lab of Prof. Yangdou Wei, Department of Biology, University of Saskatchewan
Education: Ph.D. in Biology, University of Saskatchewan, Canada. M.Sc. in Plant Pathology, Huazhong Agricultural University, China
Non-scientific Interests: swimming, traveling, rock climbing, and hiking
Brief bio:
My interest in scientific research began with an undergraduate course — General Plant Pathology at Huazhong Agricultural University, and I was attracted by various plant pathogens and their delicate parasitic mechanisms. I joined the lab of Prof. Daohong Jiang at HZAU for my M.Sc., and worked on the tritrophic interactions involving the plant rapeseed, the pathogen Sclerotinia sclerotiorum and the mycoparasite Coniothyrium minitans. After two years of office work in my hometown, I went on to pursue my Ph.D. degree in Biology in Prof. Yangdou Wei’s lab at University of Saskatchewan. My doctoral research focuses on the interactions between Arabidopsis and incompatible or compatible filamentous pathogens, involving two projects: (1) Plant biotrophic and hemibiotrophic pathogens modulate the subcellular distribution of host phosphoinositides and recruit PI(4,5)P2 as a susceptibility factor for plant disease; (2) ARP2/3 complex and formins act cooperatively to form actin patches and modulate the penetration resistance against powdery mildew invasion. I am currently working on the cellular and genetic regulation of defensive lignification in Arabidopsis in response to pathogen invasion.
姓名：覃黎
目前职位：萨斯喀彻温大学生物系，博士后
教育背景：华中农业大学，硕士；萨斯喀彻温大学，博士
兴趣爱好：游泳、旅游、攀岩和远行
个人简介：
我对科研的兴趣始于华中农业大学的一门本科课程——普通植物病理学。在学习过程中，各种植物病原体以其巧妙而独特的寄生机制深深的吸引了我。随后我加入姜道宏教授实验室攻读硕士学位，研究油菜、病原菌核盘菌和重寄生菌盾壳霉的三重营养互作。硕士毕业后，我回到美丽的家乡长阳，在农业局工作了两年，然后我决定前往加拿大萨斯喀彻温大学攻读博士学位。在卫扬斗老师的悉心指导下，我主要研究模式植物拟南芥与亲和/不亲和的丝状病原真菌之间的相互作用。一方面我们发现活体和半活体营养型植物病原菌改变了寄主细胞内磷酸肌醇的分布，并利用寄主的磷脂酰肌醇4,5-二磷酸【PI(4,5)P2】作为感病因子来促进病害发展。另一方面，我们发现肌动蛋白调节因子ARP2/3复合物和formins二者协同作用，在白粉菌侵入位点打上补丁来阻止其穿透。目前我正在研究木质化（lignification）在拟南芥抵抗病菌入侵过程中的细胞和遗传调控机理。
